Labels:text | screenshot | black and white | font | black | document OCR: syndrome Biglieri and colleagues have described a sent in the where defective 17ct-hydroxylation is p evels of adrenals and gonads. The resultant low cortisol, acting via the negative feedback mechanism, stimulate ACTH release, w essive consequent bilateral hyperplasia and ex icosterone, secretion of corticosterone and deoxyco potassium These, in turn, cause sodium retention, lkalosis. loss, hypertension, and a hypokalaemic in in men The absence of normal androgen secreti hile causes male pseudohermaphroditism, results in defective oestrogen secretion in womer Ilso primary amenorrhoea. Renin levels are suppressed,
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